Vishal Madan's Basal Cell Carcinoma PDF

By Vishal Madan

ISBN-10: 9535103091

ISBN-13: 9789535103097

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J. (1995). Etiology and pathogenesis of basal cell carcinoma. ; Fryer, A. C. (1996). Susceptibility to multiple cutaneous basal cell carcinomas: significant interactions between glutathione S-transferase GSTM1 genotypes, skin type and male gender. ; Strange, R. & Fryer; A. (1997). Basal cell carcinoma. Risk factors for basal cell carcinoma in the UK: casecontrol study in 806 patients. ; Kaaman, T. (1998). Multiple basal cell carcinomas. A clinical evaluation of risk factors. Acta Derm Venereol 78: 127-129 [31] Fusenig, N.

Verification with BCCs is yet to follow. Examination of RAR receptors (retinoic acid receptors) and RXR receptors (retinoid X receptors) on BCCs might indicate why most studies do not show any differences between therapy and placebo groups. Using immunohistochemical methods, Kamradt and Reichrath (226) investigated expression of RAR receptors in frozen sections of 15 basal cell carcinomas: RAR was most evident (3 +), RAR a little weaker (2 +) and RAR scanty (0 to 1 +). This corroborates studies by Hartmann et al.

2 Marker Ki-67 in BCCs and BCC-free tumor margin tissues in comparison with p53 Some authors compared proof of p53 with expression of the proliferation marker Ki-67. Healy et al. (163) proved 71 BCC tissues immunohistochemically with p53 and Ki-67, 17 BCCs group 1 (patients without recurrence), 17 BCCs group 2-0 (patients who relapsed some time later) and 17 BCC relapses (group 2-R). p53 was demonstrated in 95% of each group. 009. Barrett et al. (158) examined expression of p53, Ki-67 and PCNA in 27 basal cell carcinomas and compared them to histopathological BCC types.

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Basal Cell Carcinoma by Vishal Madan

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